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In liver disease models linked to metabolic syndrome, Lip-1 reduces hepatic steatosis and mitochondrial ROS dysfunction, thereby slowing disease progression (52)

Clin Endocrinol (2021) 95:46977

Preclinical models demonstrate that these compounds reduce reactive oxygen species, stabilize mitochondrial membrane potential, enhance mitophagy, and activate NRF2 dependent transcription, which translates into attenuation of ASD like behaviors in VPA, propionic acid, maternal immune activation, and BTBR paradigms (De Mattos et al., 2020

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Historical research on GHK-Cu's biological functions The review consolidates findings from laboratory studies, animal models, and human clinical trials spanning from the 1980s to 2018

Using both provides redundant angiogenic stimulation through multiple pathways