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The study revealed that the combination of apatinib and olaparib effectively suppressed ferroptosis in ovarian cancer cells via the p53-mediated Nrf2 pathway (Kang et al., 2019)
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For thoracic spine injuries involving damaged ligaments or the thoracolumbar fascia, this accelerated fibroblast activity could translate into faster and potentially higher-quality tissue repair
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Figure 1 Ferroptosis, a recently identified form of programmed cell death, is characterized by elevated levels of reactive oxygen species (ROS) and iron-dependent lipid peroxidation ( + to Fe + occurs after cellular iron uptake through transferrin receptor 1 (TFR1)
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