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These successful clinical translations demonstrate the extensive potential of ASOs, which function by causing specific mRNA degradation, exon skipping, or exon retention at either the mature or pre-mRNA level to address diverse disorders [76]

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With each cell division, telomeres lose approximately 50200 base pairs of their length

Notably, the latter also may harbor PRKAR1A aberrations [21] or loss of heterozygosity [22]

The answer is closer to yes than you think and even they might not know it