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In the real world, a drug that keeps patients on treatment accrues more cumulative benefit than a stronger drug that patients abandon

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In a murine myocardial infarction model, miR-92a inhibition improved endothelial cell function and postinfarction recovery by modulating autophagy-related genes in an endothelium-specific manner

10.1021/jacs.9b03623 79 ZhaoB.ZhangQ.LiZ

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A convincing GHK-Cu trial would need: GHK-Cu monotherapy , not a combination product, so the effect can be attributed to the peptide