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and Gonzlez, N

Nat Neurosci 6 : 11621168

Finally, as both increasing age and germ-free conditions have been associated with a decline Drosophila antioxidant responses 22,23 , we defined the requirement of the antioxidant response system in our model and find, in agreement with vertebrate studies, that genetic and pharmacologic manipulations of antioxidant response pathways results in altered sensitivity to APAP toxicity 24,25

(2018) noted that BPC-157 "counteracts dopamine system disturbances" in rodent models, producing effects that overlap, in part, with anxiolytic and neuroprotective profiles [1]