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Doses of 200500 g in rodent models produce significant healing acceleration

Unlike synthetic Wnt agonists, which can create unwanted off-target stimulation, the tripeptide appears to nudge Wnt signaling within a biological operating range

It is hypothesized to be linked to excessive MMP-1 activity, which could temporarily break down existing collagen faster than new collagen is built

Option one: switch to maintenance frequency and continue

The foundation of research backing GHK-Cus efficacy is strong, its mechanisms of action are sound, and the safety profile works in the peptides favor