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Free methionine supplementation limits alcohol-induced liver damage in rats

As discussed above, utilization of Met as a source of disulfide reducing power in TrxR1/Gsr-null hepatocytes does not consume cellular NADPH

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[84] demonstrated that in colorectal cancer, suppression of the MondoATXNIP axis promotes glucose uptake and glycolysis, leading to the induction of highly glycolytic, Th17-like Tregs

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95% CI: 0.440.81