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This mechanistic complementarity leads to a more efficient wound healing cascade, facilitating rapid closure and organized tissue reconstruction under experimental conditions

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Martinez 6,7 , Maria Galn 8,9* , Affiliation: 1 Group of Endocrinology, Diabetes and Nutrition, Institut de Recerca Sant Pau, 08041 Barcelona, Spain 2 CIBER of Diabetes and Associated Metabolic Diseases (CIBERDEM), Instituto de Salud Carlos III, 28029 Madrid, Spain Email: [email protected] ORCID: Josep Julve 1,2* Introduction Metabolic dysfunction-associated steatotic liver disease (MASLD), formerly known as non-alcoholic fatty liver disease (NAFLD), is a complex chronic metabolic dysfunction of the liver influenced by both genetic and non-genetic factors [1]

In the key trial supporting its approval for postherpetic neuralgia, somnolence was reported in 21% of patients receiving gabapentin 1,800 mg/day versus 5% on placebo [3]

Research on rat models of alkali chemical burns reveals that BPC-157 can accelerate wound closure by increasing rates of granulation tissue formation, collagen deposition, blood vessel formation, and dermal remodeling

Topical applications appear generally well-tolerated in small studies, but systemic safety, long-term effects, and drug interactions remain inadequately characterized