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Although no single gene alteration has been found that explains all the features of PWS, several unique translocation and deletion patients have narrowed a key region to explain much of the PWS phenotype to the HBII-85 snoRNA gene (reviewed in Sahoo et al )
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These studies indicate that overproduction of glutathione drives the survival and growth of a small subset of lymphoid tumor cells in 3D gels and xenografts, likely TIC or TIC-like cells, and that glutathione is selectively required for tumor growth in females of a mouse model of lymphoma
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