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In addition, dichloroaceetate treatment alone caused ER stress to an extent that was nearly as robust as the bona fide ER stressor TM, as seen by upregulation of UPR target genes (Figure 7B) and splicing of Xbp1 (Figure 7C)

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To explore the neurogenic potential of hGMSCs, cells were treated with the AEDG peptide at a concentration of 0.01 g/mL for seven days

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