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Mitochondrial metabolic reprogramming in CRC: mechanisms of therapy resistance and emerging interventions As discussed above, CRC cells adapt to hypoxia, nutrient deprivation, and therapeutic stress through mitochondrial metabolic reprogramming, which maintains redox homeostasis and organelle quality control to promote survival

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They found that the knockdown of endogenous IRS-1 significantly decreased PI3K/AKT signaling and this effect was similarly shown in BC cells with miR-148a or miR-152 overexpression leading to the inhibition of cell proliferation

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That is a risk-benefit ratio no ethical physician would accept for an elective cosmetic procedure