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Even when fertilization is achieved, oxidative stress (inflammation)-induced apoptosis (cell death) may result in embryo fragmentation implantation failure insufficient luteal hormonal levels miscarriage impaired placenta development inadequate growth of the uterine lining (necessary for embryo development congenital abnormalities More reproductive condition-specific

In Parkinsons disease, treatment with vitamin K2 reduces the formation of -synuclein fibrils and downregulates inducible nitric oxide synthase, thereby decreasing neuroinflammation and apoptosis

Although PDs etiology remains largely enigmatic, evidence implicates genetic mutations, environmental toxins, oxidative stress, mitochondrial impairment, and neuroinflammation in its pathogenesis (Madiha et al., 2021)

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Gut 58 , 15971605 (2009)