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For example, childhood trauma, stress, and exposure to toxins can affect the expression of the MAOA gene, leading to changes in MAO-A levels.[ref] During childhood and early adolescence, brain structures are rapidly developing

As direct consequence, some SnCE1 acetylation sites are enzymatically deacetylated upon exposure in the host cell following injection of SnCE1 while others might remain stable as they are not accessible for enzymatic deacetylation

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