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Research shows that females are potentially more susceptible to T2D with chronic refined carbohydrate intake, whereas males may be more susceptible to chronic disease when chronically consuming high-fat diets, particularly those high in animal products [121, 122]

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Together, these findings suggest that VRK2-deficient PC cells exhibit a metabolic preference for GSH dependency, implying that VRK2 has the potential to regulate GSH biosynthesis