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By sequestering actin monomers, the peptide facilitates rapid cell migration and survival

Antibody targeting [197] or knock-down of TME-expressed EphA3 [198] decreased angiogenesis and tumour growth

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Despite the development of numerous drugs targeting specific pathological features of AD, such as amyloid-beta (A) deposition and neurofibrillary tangles, very few have been approved in the past 20 years, underscoring the limitations of single-target therapeutic approaches