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l-carnitine treatment also was able to restore the age-related changes in the functions of inflammatory cells, and presented a protective role in the tissue destruction in inflammation by decreasing the superoxide anion production [82]

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Consistent with our characterization of cytoplasmic condensation as a cell death phenotype, cytoplasmic condensation and fluorescence of C11-BODIPY also occurred similarly to cells without -tocopherol pretreatment (Fig