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2013;1:e52715

Curr Eye Res 2021;46:47081 7

Pathogenesis and diagnosis of growth hormone deficiency in adults

The proposed mechanism involves controlling glutamate release in the synaptic cleft, modulating NMDA receptor stimulation, affecting excitatory amino acid transporter (EAAT)-mediated reuptake, and maintaining the glutamate/glutamine cycle within astrocytes

This terminology is not used much nowadays

While slight reductions in absolute muscle mass were observed, concerns about loss-associated strength declines were addressed by observations of improved power-to-weight ratios and mobility gains in mice, alongside preserved strength in the small human cohort