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Key molecular mechanisms include mitochondrial reverse electron transport activation induced by mutant proto-oncogenes KRAS/BRAF [385], oxidative inactivation of DNA repair enzymes leading to mutation accumulation [386], and constitutive activation of NRF2 pathway [387]

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But there is profit in keeping people dependent on insulin injections, metformin, and other drugs that often worsen long-term health

studies examining GH effects on IGF-1 without hypothalamic-adrenal axis confounding