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2019), while impaired gastric blood flow, oxidant damage, apoptosis, production of reactive oxygen metabolites and pro-inflammatory cytokines (e.g., tumor necrosis factor (TNF)-, interleukin (IL)-6 and IL-1) along with alterations in the expressions of nuclear factor kappa B (NF-B) and prostaglandins (PGI2 and PGE2) aggravate gastric injury (Arabaci Tamer et al