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In contrast, after the addition of SB431542, a TGF signalling inhibitor, to the high-glucose medium, -SMA expression in PSCs decreased to a nearly undetectable level

Results clearly present the chemical potentiation of LASSS on physiological functions of HGF, as revealed by enhanced receptor-binding affinity and nitration-dysfunction resistance through interaction/reaction with LASSS under physiological conditions

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The biochemical and structural characteristics of BPC-157 are determined by the peptide itself rather than its formulation format